@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix schem: . @prefix go: . @prefix do: . @prefix occursIn: . @prefix obo: . @prefix hasAnnotation: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: do:1936, obo:CL_0000115, obo:UBERON_0001637; hasAnnotation: sub:_2; rdf:object go:0008632; rdf:predicate belv:increases; rdf:subject schem:Oxidized%20Low%20Density%20Lipoprotein; a rdf:Statement . sub:_2 dct:subject "TextLocation"; rdf:value "Review" . sub:assertion rdfs:label "a(SCHEM:\"Oxidized Low Density Lipoprotein\") -> bp(GOBP:\"apoptotic process\")" . } sub:provenance { beldoc: dce:description "Approximately 2000 hand curated statements drawn from 57 PubMeds."; dce:rights "Copyright (c) 2011-2012, Selventa. All Rights Reserved."; dce:title "BEL Framework Small Corpus Document"; dct:license "Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value """One such risk factor is oxLDL, which exert pro-apoptotic effects on a variety of cell lineages, including endothelial cells and VSMCs, both in cultured cells and in arteries [19–26]. Increased formation of oxygen radicals (and other radical species) facilitates LDL oxidation and influences oxidation-sensitive mechanisms [15]."""; prov:wasQuotedFrom pubmed:12928037 . sub:_4 rdfs:comment "support@belframework.org"; rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12928037; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:31:14.763+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }