@prefix dc: . @prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix pfh: . @prefix proteinModification: . @prefix psimod: . @prefix mesh: . @prefix occursIn: . @prefix hasAnnotation: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:5464; a Protein: . sub:_2 belv:variantOf pfh:AKT%20Family; a proteinModification:, psimod:00696 . sub:_3 occursIn: mesh:D002289, species:9606; hasAnnotation: sub:_4; rdf:object sub:_2; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:_4 dc:subject "TextLocation"; rdf:value "Results" . sub:assertion rdfs:label "p(HGNC:IGF1) -> p(PFH:\"AKT Family\",pmod(P,S,473))" . } sub:provenance { beldoc: dce:description "Approximately 2000 hand curated statements drawn from 57 PubMeds."; dce:rights "Copyright (c) 2011-2012, Selventa. All Rights Reserved."; dce:title "BEL Framework Small Corpus Document"; dc:license "Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License"; pav:authoredBy sub:_6; pav:version "1.6" . sub:_5 prov:value """In all 10 cell lines tested, IGF-I increased S473 phosphorylation irrespective of endogenous levels, indicating that NSCLC cell lines phosphorylate Akt/PKB in response to IGF-I, and that the IGF-IR signaling pathway that leads to Akt/PKB activation is intact, even in cells that exhibit no Akt/PKB phosphorylation (H1355)."""; prov:wasQuotedFrom pubmed:11358816 . sub:_6 rdfs:comment "support@belframework.org"; rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:11358816; prov:wasDerivedFrom beldoc:, sub:_5 . } sub:pubinfo { this: dc:created "2014-07-03T14:29:31.484+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }