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http://amigo.geneontology.org/amigo/term/GO:0007160
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http://www.tkuhn.ch/bel2nanopub/RALc4oNB_es7G0TCvcmSZMr1ZJ39gT_DiKJsHFL3laPQQ#_4
http://purl.obolibrary.org/obo/BFO_0000066
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TextLocation
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Results
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p(HGNC:ITGB1) -> (p(HGNC:VEGFA) -> bp(GOBP:"cell-matrix adhesion"))
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p(HGNC:VEGFA) -> bp(GOBP:"cell-matrix adhesion")
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http://purl.org/dc/elements/1.1/description
Approximately 2000 hand curated statements drawn from 57 PubMeds.
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Copyright (c) 2011-2012, Selventa. All Rights Reserved.
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http://purl.org/dc/elements/1.1/title
BEL Framework Small Corpus Document
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Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License
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As shown in Fig. 3,d–e, incubation with the PKC inhibitor BIM I (as well as with the
β1-neutralizing mAb) blocked dose-dependent VEGF-induced cell adhesion to fibronectin.
This involvement of PKC in MM cell adhesion to fibronectin was further
confirmed by a dose-dependent increment of adhesion triggered by PMA stimulation
(Fig. 3d) similar to that induced by VEGF. Taken together, our results show that
VEGF transiently enhances MM cell adhesion to fibronectin, dependent on both
PKC and β1 integrin activity.
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http://www.ncbi.nlm.nih.gov/pubmed/11751905
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support@belframework.org
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Selventa
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2014-07-03T14:31:16.249+02:00
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