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http://www.tkuhn.ch/bel2nanopub/RARjESsBYXh6WSoYw6qZHU-Os7FHGSeizNHl7EGu4fiks#_1
http://semanticscience.org/resource/SIO_000139
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0016301
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=3942
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
http://www.tkuhn.ch/bel2nanopub/RARjESsBYXh6WSoYw6qZHU-Os7FHGSeizNHl7EGu4fiks#_3
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http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=6081
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
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http://www.w3.org/1999/02/22-rdf-syntax-ns#predicate
http://www.selventa.com/vocabulary/increases
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http://www.w3.org/1999/02/22-rdf-syntax-ns#Statement
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http://purl.obolibrary.org/obo/BFO_0000066
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http://purl.obolibrary.org/obo/BFO_0000066
http://www.lgcstandards-atcc.org/Products/All/CRL-2057.aspx
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http://purl.obolibrary.org/obo/BFO_0000066
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http://semanticscience.org/resource/SIO_000255
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http://www.selventa.com/vocabulary/increases
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TextLocation
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Results
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a(CHEBI:glucose) -> p(HGNC:INS)
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http://www.w3.org/2000/01/rdf-schema#label
kin(p(HGNC:MTOR)) -> (a(CHEBI:glucose) -> p(HGNC:INS))
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http://purl.org/dc/elements/1.1/description
Approximately 2000 hand curated statements drawn from 57 PubMeds.
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Copyright (c) 2011-2012, Selventa. All Rights Reserved.
http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel
http://purl.org/dc/elements/1.1/title
BEL Framework Small Corpus Document
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Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License
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1.6
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we combined the stimulation with glucose or insulin with the cotreatment with inhibitors
of the insulin receptor tyrosine kinase [hydroxy-2-naphthalenylmethylphosphonic
acid trisacetoxymethyl ester (HNMPA)], PI 3-kinase (wortmannin and LY294002),
and mTOR (rapamycin). As shown in Figs. 1 and 2, HNMPA, wortmannin, LY294002,
and rapamycin inhibited both glucose- and insulin-stimulated insulin biosynthesis
in both human pancreatic islets and RIN β-cells.
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http://www.w3.org/ns/prov#wasQuotedFrom
http://www.ncbi.nlm.nih.gov/pubmed/15001544
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support@belframework.org
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Selventa
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2014-07-03T14:29:33.044+02:00
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