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http://www.tkuhn.ch/bel2nanopub/RAX0b2a9RNuD8_Yefa83OjCj-i6hPjah4CKXPKzi6vR2U#_1
http://purl.obolibrary.org/obo/RO_0002204
http://resource.belframework.org/belframework/1.0/namespace/selventa-named-human-protein-families/VEGF%20Family
http://www.tkuhn.ch/bel2nanopub/RAX0b2a9RNuD8_Yefa83OjCj-i6hPjah4CKXPKzi6vR2U#_1
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http://www.tkuhn.ch/bel2nanopub/RAX0b2a9RNuD8_Yefa83OjCj-i6hPjah4CKXPKzi6vR2U#_2
http://semanticscience.org/resource/SIO_000139
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http://amigo.geneontology.org/amigo/term/GO:0005525
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=659
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAX0b2a9RNuD8_Yefa83OjCj-i6hPjah4CKXPKzi6vR2U#_4
http://purl.obolibrary.org/obo/BFO_0000066
http://purl.bioontology.org/ontology/MSH/D021941
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http://purl.bioontology.org/ontology/MSH/D042783
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http://www.selventa.com/vocabulary/increases
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http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
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http://www.w3.org/2000/01/rdf-schema#label
p(PFH:"VEGF Family") -> gtp(p(HGNC:ARF6))
http://www.tkuhn.ch/bel2nanopub/RAX0b2a9RNuD8_Yefa83OjCj-i6hPjah4CKXPKzi6vR2U#provenance
http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel
http://purl.org/dc/elements/1.1/description
Approximately 2000 hand curated statements drawn from 57 PubMeds.
http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel
http://purl.org/dc/elements/1.1/rights
Copyright (c) 2011-2012, Selventa. All Rights Reserved.
http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel
http://purl.org/dc/elements/1.1/title
BEL Framework Small Corpus Document
http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel
http://purl.org/dc/terms/license
Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License
http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel
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1.6
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http://www.w3.org/ns/prov#value
In ECs, VEGF stimulates ARF6 association with Rac1, which is cotemporaneous with Rac1
activation. The dominant negative ARF6 (T27N),inhibits VEGF-stimulated GTP-loading
of Rac1, ROS production, and VEGFR2 autophosphorylation. These results suggest
that ARF6 is an upstream mediator for Rac1, which may contribute to ROS production
that mediates VEGFR2 autophosphorylation in ECs (Fig. 1).
http://www.tkuhn.ch/bel2nanopub/RAX0b2a9RNuD8_Yefa83OjCj-i6hPjah4CKXPKzi6vR2U#_5
http://www.w3.org/ns/prov#wasQuotedFrom
http://www.ncbi.nlm.nih.gov/pubmed/17511588
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support@belframework.org
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Selventa
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http://www.ncbi.nlm.nih.gov/pubmed/17511588
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http://www.tkuhn.ch/bel2nanopub/RAX0b2a9RNuD8_Yefa83OjCj-i6hPjah4CKXPKzi6vR2U
http://purl.org/dc/terms/created
2014-07-03T14:29:33.924+02:00
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http://orcid.org/0000-0001-6818-334X
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http://orcid.org/0000-0002-1267-0234