@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix go: . @prefix hasAnnotation: . @prefix species: . @prefix occursIn: . @prefix obo: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:2444554; a Protein: . sub:_2 occursIn: obo:CL_0000057, species:10090; hasAnnotation: sub:_3; rdf:object go:0008632; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:_3 dct:subject "TextLocation"; rdf:value "Results" . sub:assertion rdfs:label "p(MGI:Mapkap1) -| bp(GOBP:\"apoptotic process\")" . } sub:provenance { beldoc: dce:description "Approximately 2000 hand curated statements drawn from 57 PubMeds."; dce:rights "Copyright (c) 2011-2012, Selventa. All Rights Reserved."; dce:title "BEL Framework Small Corpus Document"; dct:license "Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value """In etoposide-treated cells, there was a 2-fold increase in the number of cells that underwent apoptosis after 48 hr in SIN1 knockouts (Figure 4D). Thus, we find that the SIN1−/− cells were more sensitive to stress-induced apoptosis, suggesting that Akt-Ser473 phosphorylation plays an important role in cell survival (Figures 4C and 4D)."""; prov:wasQuotedFrom pubmed:16962653 . sub:_5 rdfs:comment "support@belframework.org"; rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:16962653; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:31:15.272+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }