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TextLocation
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Review
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p(PFH:"IL1 Family") -> complex(p(HGNC:MYD88),p(HGNC:IRAK1))
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Approximately 2000 hand curated statements drawn from 57 PubMeds.
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Copyright (c) 2011-2012, Selventa. All Rights Reserved.
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BEL Framework Small Corpus Document
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Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License
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Although the kinase activity of IRAK-1 increases strongly following IL-1 stimulation,
IRAK-1 kinase activity is not required for its signaling function, because
overexpression of a kinase-defective mutant of IRAK-1 is observed to strongly induce
NF-κB activation in cells otherwise deficient for IRAK-1. Upon stimulation,
IRAK-1 is recruited to the receptor through a homophilic interaction with the DD
of MyD88.
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support@belframework.org
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Selventa
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2014-07-03T14:29:33.156+02:00
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