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TextLocation
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Results
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tscript(p(MGI:Ctnnb1)) => r(MGI:Bmp4)
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Approximately 2000 hand curated statements drawn from 57 PubMeds.
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Copyright (c) 2011-2012, Selventa. All Rights Reserved.
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BEL Framework Small Corpus Document
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Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License
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Co-transfection of luciferase reporter plasmids containing these sequences along with
expression plasmids encoding either an activated β-catenin containing mutations
in the GSK-3h phosphorylation sites (β-catenin/4145) or with a TCF3-VP16 fusion
protein demonstrated that both promoter sequences could be activated by transcriptional
mediators of Wnt signaling (Figs. 7A and B). Addition of both h-catenin/4154
and TCF3-VP16 resulted in cooperative activation of both promoters (Figs.
7A and B). Furthermore, a dominantnegative TCF3 construct repressed both BMP4
and N-myc promoters (Figs. 7C and D). Chromatin immunoprecipitation assays show
that β-catenin associates with the conserved LEF/TCF DNA binding sites located
in the BMP4 and N-myc promoters, indicating direct regulation of these promoters
by canonical Wnt signaling (Figs. 7E and F). Together, these results demonstrate
that BMP4 and Nmyc are targets of Wnt/β-catenin signaling in lung airway epithelium.
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2014-07-03T14:29:32.114+02:00
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