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Results
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a(CHEBI:glucosamine) -> p(RGD:Irs1,pmod(P,S,612))
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Approximately 2000 hand curated statements drawn from 57 PubMeds.
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Copyright (c) 2011-2012, Selventa. All Rights Reserved.
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BEL Framework Small Corpus Document
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Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License
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Exposure of RIN β-cells to glucosamine increased phosphorylation of both IRS-1 Ser307
and IRS-1 Ser612, which was reversed by treatment with either a cell-permeable
JNK inhibitor or PD98059, a reversible MAPK kinase (MEK)1 inhibitor, the enzyme
that directly activates ERK1/2, respectively (Fig. 10, A and B)
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2014-07-03T14:29:33.105+02:00
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