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an alternative pathway for phosphatidylcholine synthesis, which is quantitatively only in the liver, is the methylation of phosphatidylethanolamine to phosphatidylcholine via phosphatidylethanolamine N-methyltransferase, Pemt mice with methyltransferase gene disrupted have greatly reduced plasma levels of triacylglycerol and APOB100, but not APOB48 secretion of APOB100 and triacylglycerols from hepatocytes derived from these mice is simiarly impaired phosphatidylcholine synthesis is required for normal APOB secretion
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