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All Rights Reserved. http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Small Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel http://purl.org/dc/terms/license Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAqeAwIaSg5JnrqtkHeyExFQtuYKI9KHRpHJP1SWKjKxY#_6 http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel http://purl.org/pav/version 1.6 http://www.tkuhn.ch/bel2nanopub/RAqeAwIaSg5JnrqtkHeyExFQtuYKI9KHRpHJP1SWKjKxY#_5 http://www.w3.org/ns/prov#value To identify a function that could be linked specifically to Akt-Ser473 phosphorylation, we further examined known Akt substrates that may have defective phosphorylation in SIN1−/− cells. We found that phosphorylation of FoxO1/3a (also called FKHR/FKHRL1) (Greer and Brunet, 2005), was affected in SIN1−/− cells. 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