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All Rights Reserved. http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Small Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel http://purl.org/dc/terms/license Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RA_hHn7BliFJIy5SyAy_gOP4xhvYNdwA_zllI2SRN2c7w#_5 http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RA_hHn7BliFJIy5SyAy_gOP4xhvYNdwA_zllI2SRN2c7w#_4 http://www.w3.org/ns/prov#value As illustrated in Fig.2, A–C, the expression of E-selectin correlated with an increased adhesion of both colon carcinoma HT-29 cells and HL-60 leukemia cells to a monolayer of HUVEC. After 30 min, the number of HT-29 cells that adhered to HUVEC-expressing E-selectin, following activation with TNFα, was 5-fold higher than when adhering to inactivated HUVEC. Similarly, HT-29 cells quickly adhered to immobilized recombinant human E-selectin/Fc chimera (data not shown). 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