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http://www.tkuhn.ch/bel2nanopub/RAyCkHcclQ_rgkfWorUVUXmKF6IJTLFSPP2e1WProTxP8#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RAyCkHcclQ_rgkfWorUVUXmKF6IJTLFSPP2e1WProTxP8#_4
http://purl.obolibrary.org/obo/BFO_0000066
http://purl.obolibrary.org/obo/EFO_0001200
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TextLocation
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Abstract
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http://www.w3.org/2000/01/rdf-schema#label
tscript(p(HGNC:USF1)) causesNoChange r(HGNC:IGF2R)
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http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel
http://purl.org/dc/elements/1.1/description
Approximately 2000 hand curated statements drawn from 57 PubMeds.
http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel
http://purl.org/dc/elements/1.1/rights
Copyright (c) 2011-2012, Selventa. All Rights Reserved.
http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel
http://purl.org/dc/elements/1.1/title
BEL Framework Small Corpus Document
http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel
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Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License
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USF1 and USF2 bound the IGF2R promoter in vitro, and both USF1 and USF2, but not c-Myc,
were present within the IGF2R promoter-associated chromatin in vivo. Overexpressed
USF2, but not USF1, transactivated the IGF2R promoter, and IGF2R mRNA
was markedly decreased by expression of a USF-specific dominant negative mutant,
identifying IGF2R as a USF2 target.
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http://www.ncbi.nlm.nih.gov/pubmed/12857727
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support@belframework.org
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Selventa
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2014-07-03T14:31:16.985+02:00
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