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TextLocation
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Approximately 2000 hand curated statements drawn from 57 PubMeds.
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Copyright (c) 2011-2012, Selventa. All Rights Reserved.
http://resource.belframework.org/belframework/1.0/knowledge/small_corpus.bel
http://purl.org/dc/elements/1.1/title
BEL Framework Small Corpus Document
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Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License
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The β-casein promoter, known to be induced by Stat5 when activated by prolactin via
prolactin receptors and JAK2, was next used to test the function of the phosphorylated
Stat5. T47D-YB cells were pretreated with either R5020 or ethanol vehicle
for 48 h. Cells were then washed, transfected with a β-casein promoter-luciferase
reporter, treated with either R5020 or prolactin, and harvested for luciferase
assay 24 h after hormone treatment. We found that in T47D-YB breast cancer
cells, prolactin alone has minimal to no significant effect on the β-casein luciferase
promoter. However, after R5020 pretreatment, a 6–7-fold induction of the
β-casein luciferase promoter was observed (Fig.7B). Thus R5020 sensitized these
breast cancer cells to the transcriptional effects of prolactin mediated through
Stat5.
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http://www.ncbi.nlm.nih.gov/pubmed/9813040
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support@belframework.org
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Selventa
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2014-07-03T14:29:30.811+02:00
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